From: To: Jeffrey Epstein <[email protected]> Subject: Fwd: TSH I Date: Thu, 11 Jul 2013 14:56:39 +0000 Forwarded message From: Tohru Date: Tue, Jul 9, 2013 at 1:15 AM Sub'ect: RE: TSH1 To: Dear Christina; I am answering your questions. (1) TSH1 is name of our brain food. This componenet is ferulic acid. But I am sorry I can't open the other important component of TSH1 at present. (2) Since my research made clear that the pathogen of Alzheimer's disease was homocysteic acid in blood, then first my TSH1 first blocks the homocysteic acid in brain at NMDA receptor. Second my TSH1 decopomses homocysteic acid, especially sulfide group of homocysteic acid in blood. My TSH1 stimulates the urinary excretion of homocysteic acid in blood. Alzheimer's and late onset of Alzheimer's disease are same pathogen. You may question that then why late onset of Alzheimer's and early onset of Alzheimer's disease are different in onset. That is why amyloid protein is involved in onset. Early onset of Alzheimer's disease shows high level of amyloid protein, which increases homocysteic acid toxicity. Then lower level of homocysteic acid induced the onset of Alzheimer's. It is important that amyloid protein only is not a pathogen of Alzheimer's disease. (3) TSH1 is orally administrated. 2g of TSH1 was administrated. (4) Basically ferulic acid is already known in Japan as a potent therapy effect of Alzheimer's disease. However ferulic acid efficacy is rather small. That is 40% of ferulic acid amnistrated patients showed the recovery. My TSH1 clinical trail was Phase 2 . Phase 1 was already shown no toxicity of TSH1. (5) !00% of all TSH1 adminstrated patients showed the cognitive recovery EFTA00703251